Authors

Jennifer W Leiding
Tiphanie P Vogel
Valentine G J Santarlas
Rahul Mhaskar
Madison R Smith
Alexandre Carisey
Alexander Vargas-Hernández
Manuel Silva-Carmona
Maximilian Heeg
Anne Rensing-Ehl
Bénédicte Neven
Jérôme Hadjadj
Sophie Hambleton
Timothy Ronan Leahy
Kornvalee Meesilpavikai
Charlotte Cunningham-Rundles
Cullen M Dutmer
Svetlana O Sharapova
Mervi Taskinen
Ignatius Chua
Rosie Hague
Christian Klemann
Larysa Kostyuchenko
Tomohiro Morio
Akaluck Thatayatikom
Ahmet Ozen
Anna Scherbina
Cindy S Bauer
Sarah E Flanagan
Eleonora Gambineri
Lisa Giovannini-Chami
Jennifer Heimall
Kathleen E Sullivan
Eric Allenspach
Neil Romberg
Sean G Deane
Benjamin T Prince
Melissa J Rose
John Bohnsack
Talal Mousallem
Rohith Jesudas
Maria Marluce Dos Santos Vilela
Michael O'Sullivan
Jana Pachlopnik Schmid
Štěpánka Průhová
Adam Klocperk
Matthew Rees
Helen Su
Sami Bahna
Safa Baris
Lisa M Bartnikas
Amy Chang Berger
Tracy A Briggs
Shannon Brothers
Vanessa Bundy
Alice Y Chan
Shanmuganathan Chandrakasan
Mette Christiansen
Theresa Cole
Matthew C Cook
Mukesh M Desai
Ute Fischer
David A Fulcher
Silvanna Gallo
Amelie Gauthier
Andrew R Gennery
José Gonçalo Marques
Frédéric Gottrand
Bodo Grimbacher
Eyal Grunebaum
Emma Haapaniemi
Sari Hämäläinen
Kaarina Heiskanen
Tarja Heiskanen-Kosma
Hal M Hoffman
Luis Ignacio Gonzalez-Granado
Anthony L Guerrerio
Leena Kainulainen
Ashish Kumar
Monica G Lawrence
Carina Levin
Timi Martelius
Olaf Neth
Peter Olbrich
Alejandro Palma
Niraj C Patel
Tamara Pozos
Kahn Preece
Saúl Oswaldo Lugo Reyes
Mark A Russell
Yael Schejter
Christine Seroogy
Jan Sinclair
Effie Skevofilax
Daniel Suan
Daniel Suez
Paul Szabolcs
Helena Velasco
Klaus Warnatz
Kelly Walkovich
Austen Worth
STAT3 GOF Working Group Members
Mikko R J Seppänen
Troy R Torgerson
Georgios Sogkas
Stephan Ehl
Stuart G Tangye
Megan A Cooper
Joshua D Milner
Lisa R Forbes Satter

Publication Date

4-1-2023

Journal

The Journal of Allergy and Clinical Immunology

DOI

10.1016/j.jaci.2022.09.002

PMID

36228738

PMCID

PMC10081938

PubMedCentral® Posted Date

4-1-2024

PubMedCentral® Full Text Version

Author MSS

Published Open-Access

yes

Keywords

Child, Humans, Autoimmunity, Cohort Studies, Gain of Function Mutation, Immune System Diseases, Immunologic Deficiency Syndromes, Mutation, STAT3 Transcription Factor, Cell Proliferation, Lymphocytes

Abstract

BACKGROUND: In 2014, germline signal transducer and activator of transcription (STAT) 3 gain-of-function (GOF) mutations were first described to cause a novel multisystem disease of early-onset lymphoproliferation and autoimmunity.

OBJECTIVE: This pivotal cohort study defines the scope, natural history, treatment, and overall survival of a large global cohort of patients with pathogenic STAT3 GOF variants.

METHODS: We identified 191 patients from 33 countries with 72 unique mutations. Inclusion criteria included symptoms of immune dysregulation and a biochemically confirmed germline heterozygous GOF variant in STAT3.

RESULTS: Overall survival was 88%, median age at onset of symptoms was 2.3 years, and median age at diagnosis was 12 years. Immune dysregulatory features were present in all patients: lymphoproliferation was the most common manifestation (73%); increased frequencies of double-negative (CD4-CD8-) T cells were found in 83% of patients tested. Autoimmune cytopenias were the second most common clinical manifestation (67%), followed by growth delay, enteropathy, skin disease, pulmonary disease, endocrinopathy, arthritis, autoimmune hepatitis, neurologic disease, vasculopathy, renal disease, and malignancy. Infections were reported in 72% of the cohort. A cellular and humoral immunodeficiency was observed in 37% and 51% of patients, respectively. Clinical symptoms dramatically improved in patients treated with JAK inhibitors, while a variety of other immunomodulatory treatment modalities were less efficacious. Thus far, 23 patients have undergone bone marrow transplantation, with a 62% survival rate.

CONCLUSION: STAT3 GOF patients present with a wide array of immune-mediated disease including lymphoproliferation, autoimmune cytopenias, and multisystem autoimmunity. Patient care tends to be siloed, without a clear treatment strategy. Thus, early identification and prompt treatment implementation are lifesaving for STAT3 GOF syndrome.

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