Language

English

Publication Date

2-20-2025

Journal

Nature Communications

DOI

10.1038/s41467-025-56927-2

PMID

39979240

PMCID

PMC11842757

PubMedCentral® Posted Date

2-20-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Zika virus (ZIKV) is unique among orthoflaviviruses in its vertical transmission capacity in humans, yet the underlying mechanisms remain incompletely understood. Here, we show that ZIKV induces tunneling nanotubes (TNTs) in placental trophoblasts which facilitate transfer of viral particles, proteins, mitochondria, and RNA to neighboring uninfected cells. TNT formation is driven exclusively via ZIKV non-structural protein 1 (NS1). Specifically, the N-terminal 1-50 amino acids of membrane-bound ZIKV NS1 are necessary for triggering TNT formation in host cells. Trophoblasts infected with TNT-deficient ZIKV

Keywords

Trophoblasts, Zika Virus, Humans, Viral Nonstructural Proteins, Mitochondria, Zika Virus Infection, Female, Pregnancy, Animals, Nanotubes, Placenta, Infectious Disease Transmission, Vertical

Published Open-Access

yes

nihms-2026315-f0004.jpg (404 kB)
Graphical Abstract

Share

COinS
 
 

To view the content in your browser, please download Adobe Reader or, alternately,
you may Download the file to your hard drive.

NOTE: The latest versions of Adobe Reader do not support viewing PDF files within Firefox on Mac OS and if you are using a modern (Intel) Mac, there is no official plugin for viewing PDF files within the browser window.