Publication Date

5-1-2023

Journal

Kidney Medicine

DOI

10.1016/j.xkme.2023.100618

PMID

37113163

PMCID

PMC10127135

PubMedCentral® Posted Date

2-15-2023

PubMedCentral® Full Text Version

Post-print

Published Open-Access

yes

Keywords

Azithromycin, dialysate potassium, fluoroquinolones, hemodialysis, sudden cardiac death, USRDS

Abstract

RATIONALE & OBJECTIVE: Treatment with certain QT interval-prolonging antibiotics is associated with a higher risk of sudden cardiac death among individuals with hemodialysis-dependent kidney failure. Concurrent exposure to large serum-to-dialysate potassium gradients, which promote large potassium shifts, may augment the proarrhythmic effects of these medications. The primary objective of this study was to examine whether the serum-to-dialysate gradient modifies the cardiac safety of azithromycin, and separately, levofloxacin/moxifloxacin.

STUDY DESIGN: Retrospective observational cohort study using a new-user study design.

SETTING & POPULATION: Adult in-center hemodialysis patients with Medicare coverage in the US Renal Data System (2007-2017).

EXPOSURE: Initiation of azithromycin (or levofloxacin/moxifloxacin) as compared to amoxicillin-based antibiotics (

OUTCOMES: Sudden cardiac death (14 days).

ANALYTICAL APPROACH: Inverse probability of treatment-weighted survival models to estimate HRs and robust 95% CIs.

RESULTS: The azithromycin versus amoxicillin-based antibiotic cohort included 89,379 unique patients with 113,516 azithromycin and 103,493 amoxicillin-based treatment episodes. Azithromycin versus amoxicillin-based antibiotic treatment was associated with a higher risk of sudden cardiac death overall, HR, 1.68; 95% CI, 1.31-2.16. The risk was numerically higher when the baseline serum-to-dialysate potassium gradient was ≥3 mEq/L compared with/L (HR, 2.22; 95% CI, 1.46-3.40 vs HR, 1.43; 95% CI. 1.04-1.96,

LIMITATIONS: Residual confounding.

CONCLUSIONS: Although treatment with azithromycin and, separately, respiratory fluoroquinolones were each associated with a heightened risk of sudden cardiac death, this risk was augmented in the setting of larger serum-to-dialysate potassium gradients. Minimizing the potassium gradient may be an approach to reduce the cardiac risk of these antibiotics.

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Graphical Abstract

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