Authors

Konstantinos Hatzikotoulas
Lorraine Southam
Lilja Stefansdottir
Cindy G Boer
Merry-Lynn McDonald
J Patrick Pett
Young-Chan Park
Margo Tuerlings
Rick Mulders
Andrei Barysenka
Ana Luiza Arruda
Vinicius Tragante
Alison Rocco
Norbert Bittner
Shibo Chen
Susanne Horn
Vinodh Srinivasasainagendra
Ken To
Georgia Katsoula
Peter Kreitmaier
Amabel M M Tenghe
Arthur Gilly
Liubov Arbeeva
Lane G Chen
Agathe M de Pins
Daniel Dochtermann
Cecilie Henkel
Jonas Höijer
Shuji Ito
Penelope A Lind
Bitota Lukusa-Sawalena
Aye Ko Ko Minn
Marina Mola-Caminal
Akira Narita
Chelsea Nguyen
Ene Reimann
Micah D Silberstein
Anne-Heidi Skogholt
Hemant K Tiwari
Michelle S Yau
Ming Yue
Wei Zhao
Jin J Zhou
George Alexiadis
Karina Banasik
Søren Brunak
Archie Campbell
Jackson T S Cheung
Joseph Dowsett
Tariq Faquih
Jessica D Faul
Lijiang Fei
Anne Marie Fenstad
Takamitsu Funayama
Maiken E Gabrielsen
Chinatsu Gocho
Kirill Gromov
Thomas Hansen
Georgi Hudjashov
Thorvaldur Ingvarsson
Jessica S Johnson
Helgi Jonsson
Saori Kakehi
Juha Karjalainen
Elisa Kasbohm
Susanna Lemmelä
Kuang Lin
Xiaoxi Liu
Marieke Loef
Massimo Mangino
Daniel McCartney
Iona Y Millwood
Joshua Richman
Mary B Roberts
Kathleen A Ryan
Dino Samartzis
Manu Shivakumar
Søren T Skou
Sachiyo Sugimoto
Ken Suzuki
Hiroshi Takuwa
Maris Teder-Laving
Laurent Thomas
Kohei Tomizuka
Constance Turman
Stefan Weiss
Tian T Wu
Eleni Zengini
Yanfei Zhang
arcOGEN Consortium
ARGO Consortium
DBDS Genomic Consortium
Estonian Biobank Research Team
FinnGen
Genes & Health Research Team
HUNT All-In Pain
Million Veteran Program
Regeneron Genetics Center
Manuel Allen Revez Ferreira
George Babis
Aris Baras
Tyler Barker
David J Carey
Kathryn S E Cheah
Zhengming Chen
Jason Pui-Yin Cheung
Mark Daly
Renée de Mutsert
Charles B Eaton
Christian Erikstrup
Ove Nord Furnes
Yvonne M Golightly
Daniel F Gudbjartsson
Nils P Hailer
Caroline Hayward
Marc C Hochberg
Georg Homuth
Laura M Huckins
Kristian Hveem
Shiro Ikegawa
Muneaki Ishijima
Minoru Isomura
Marcus Jones
Jae H Kang
Sharon L R Kardia
Margreet Kloppenburg
Peter Kraft
Nobuyuki Kumahashi
Suguru Kuwata
Ming Ta Michael Lee
Phil H Lee
Robin Lerner
Liming Li
Steve A Lietman
Luca Lotta
Michelle K Lupton
Reedik Mägi
Nicholas G Martin
Timothy E McAlindon
Sarah E Medland
Karl Michaëlsson
Braxton D Mitchell
Dennis O Mook-Kanamori
Andrew P Morris
Toru Nabika
Fuji Nagami
Amanda E Nelson
Sisse Rye Ostrowski
Aarno Palotie
Ole Birger Pedersen
Frits R Rosendaal
Mika Sakurai-Yageta
Carsten Oliver Schmidt
Pak Chung Sham
Jasvinder A Singh
Diane T Smelser
Jennifer A Smith
You-Qiang Song
Erik Sørensen
Gen Tamiya
Yoshifumi Tamura
Chikashi Terao
Gudmar Thorleifsson
Anders Troelsen
Aspasia Tsezou
Yuji Uchio
A G Uitterlinden
Henrik Ullum
Ana M Valdes
David A van Heel
Robin G Walters
David R Weir
J Mark Wilkinson
Bendik S Winsvold
Masayuki Yamamoto
John-Anker Zwart
Kari Stefansson
Ingrid Meulenbelt
Sarah A Teichmann
Joyce B J van Meurs
Unnur Styrkarsdottir
Eleftheria Zeggini

Language

English

Publication Date

5-1-2025

Journal

Nature

DOI

10.1038/s41586-025-08771-z

PMID

40205036

PMCID

PMC12119359

PubMedCentral® Posted Date

4-9-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Osteoarthritis is the third most rapidly growing health condition associated with disability, after dementia and diabetes1. By 2050, the total number of patients with osteoarthritis is estimated to reach 1 billion worldwide2. As no disease-modifying treatments exist for osteoarthritis, a better understanding of disease aetiopathology is urgently needed. Here we perform a genome-wide association study meta-analyses across up to 489,975 cases and 1,472,094 controls, establishing 962 independent associations, 513 of which have not been previously reported. Using single-cell multiomics data, we identify signal enrichment in embryonic skeletal development pathways. We integrate orthogonal lines of evidence, including transcriptome, proteome and epigenome profiles of primary joint tissues, and implicate 700 effector genes. Within these, we find rare coding-variant burden associations with effect sizes that are consistently higher than common frequency variant associations. We highlight eight biological processes in which we find convergent involvement of multiple effector genes, including the circadian clock, glial-cell-related processes and pathways with an established role in osteoarthritis (TGFβ, FGF, WNT, BMP and retinoic acid signalling, and extracellular matrix organization). We find that 10% of the effector genes express a protein that is the target of approved drugs, offering repurposing opportunities, which can accelerate translation.

Keywords

Humans, Osteoarthritis, Genome-Wide Association Study, Genomics, Translational Research, Biomedical, Transcriptome, Male, Female, Case-Control Studies, Single-Cell Analysis, Signal Transduction, Neuroglia, Epigenome, Proteome, Osteoarthritis, Genome-wide association studies, Genomics, Functional genomics, Translational research

Published Open-Access

yes

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