Language

English

Publication Date

7-1-2025

Journal

Nature Communications

DOI

10.1038/s41467-025-60546-2

PMID

40595486

PMCID

PMC12214859

PubMedCentral® Posted Date

7-1-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Mitochondrial membrane dynamics control the shape, number, and distribution of mitochondria and regulate energy production and cell health. In a screen for yeast mutants with increased levels of templated insertions (~10-1000 bp) in the nuclear genome, we identified mitochondrial fusion deficient mutants (mgm1Δ, ugo1Δ, fzo1Δ). We found that fusion mutants activate the iron regulon, have decreased iron-sulfur clusters (ISCs), and increased DNA damage, suggesting a role of iron homeostasis in preventing insertions. Consistently, a secondary screen found mutants affecting iron-sulfur cluster production (yfh1Δ, grx5Δ), vacuolar iron storage (ccc1Δ) or general iron homeostasis (aft1Δ) to exhibit high insertion levels. Treatment with iron chelators or hydrogen peroxide also increased insertions. We propose that iron dysregulation leading to oxidative DNA damage and compromised DNA repair drives insertions. These studies suggest that severe iron imbalance, associated with many human diseases and pharmacological treatments, can trigger genome instability in the form of templated insertions.

Keywords

Iron, Saccharomyces cerevisiae, Saccharomyces cerevisiae Proteins, DNA Damage, Mitochondrial Dynamics, Mitochondria, DNA Repair, Homeostasis, Mutation, Mutagenesis, Insertional, Hydrogen Peroxide, Genomic Instability, Iron-Sulfur Proteins, Transcription Factors, Genomic instability, Mitochondria, Chromosomes

Published Open-Access

yes

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