Language

English

Publication Date

12-3-2025

Journal

Nature Communications

DOI

10.1038/s41467-025-66120-0

PMID

41339307

PMCID

PMC12675759

PubMedCentral® Posted Date

12-3-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Protein phosphorylation plays a crucial role in regulating the cytoskeletal and membrane proteins at the axon initial segment (AIS). However, our knowledge of AIS-specific kinases and phosphatases is very limited. Here, we report the identification of a protein phosphatase 2A (PP2A) B55 regulatory subunit enriched at the AIS in mice: Ppp2r2c. Our results demonstrate that PP2A-B55 subunits exhibit substantial heterogeneity in their subcellular localization and function. Notably, the Ppp2r2c subunit is selectively concentrated at the AIS, and this enrichment is driven by its unique structure. Utilizing a microelectrode array system (MEA), we show that Ppp2r2c modulates neuronal activity during in vitro development. With phosphoproteomics, we further reveal that the potassium channel Kv1.2 is one of the downstream targets that link Ppp2r2c activity to neuronal excitability. Together, these data provide a critical entry point for understanding the mechanisms of PP2A-mediated local phospho-regulation at the AIS.

Keywords

Animals, Protein Phosphatase 2, Phosphorylation, Mice, Axon Initial Segment, Kv1.2 Potassium Channel, Neurons, Axons, Mice, Inbred C57BL, Cells, Cultured, Cellular neuroscience, Molecular neuroscience

Published Open-Access

yes

Share

COinS
 
 

To view the content in your browser, please download Adobe Reader or, alternately,
you may Download the file to your hard drive.

NOTE: The latest versions of Adobe Reader do not support viewing PDF files within Firefox on Mac OS and if you are using a modern (Intel) Mac, there is no official plugin for viewing PDF files within the browser window.