Language

English

Publication Date

1-12-2025

Journal

Neuro-Oncology

DOI

10.1093/neuonc/noae193

PMID

39427326

PMCID

PMC11726257

PubMedCentral® Posted Date

10-19-2024

PubMedCentral® Full Text Version

Post-print

Abstract

The field of immunology has traditionally focused on immune checkpoint modulation of adaptive immune cells. However, many malignancies such as glioblastoma are mostly devoid of T cells and rather are enriched with immunosuppressive myeloid cells of the innate immune system. While some immune checkpoint targets are shared between adaptive and innate immunity, myeloid-specific checkpoints could also serve as potential therapeutics. To better understand the impact of immune checkpoint blockade on myeloid cells, we systematically summarize the current literature focusing on the direct immunological effects of PD-L1/PD-1, CD24/Siglec-10, collagen/LAIR-1, CX3CL1/CX3CR1, and CXCL10/CXCR3. By synthesizing the molecular mechanisms and the translational implications, we aim to prioritize agents in this category of therapeutics for glioblastoma.

Keywords

Humans, Glioblastoma, Myeloid Cells, Immune Checkpoint Inhibitors, Brain Neoplasms, Animals, Immunotherapy, glioblastoma, immune-checkpoint blockade, myeloid cells

Published Open-Access

yes

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