Language

English

Publication Date

12-15-2025

Journal

Journal of Clinical Investigation

DOI

10.1172/JCI194134

PMID

41392980

PMCID

PMC12700539

PubMedCentral® Posted Date

12-15-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Mitochondrial metabolism orchestrates T cell functions, yet the role of specific mitochondrial components in distinct T cell subsets remains poorly understood. Here, we explored the role of mitochondrial complex II (MC II), the only complex from the electron transport chain (ETC) that plays a role in both ETC and metabolism, in regulating T cell functions. Surprisingly, MC II exerts divergent effects on CD4+ and CD8+ T cell activation and function. Using T cell-specific MC II subunit, succinate dehydrogenase A-deficient (SDHA-deficient) mice, we integrated single-cell RNA-seq and metabolic profiling, with in vitro and in vivo T cell functional assays to illuminate these differences. SDHA deficiency induced metabolic changes and remodeled gene expression exclusively in activated T cells. In CD4+ T cells, SDHA loss dampened both oxidative phosphorylation (OXPHOS) and glycolysis, impaired cytokine production, proliferation, and reduced CD4+ T cell-mediated graft-versus-host disease after allogeneic stem cell transplantation (SCT). In contrast, SDHA deficiency in CD8+ T cells reduced OXPHOS but paradoxically upregulated glycolysis and demonstrated enhanced cytotoxic functions in vitro and in vivo. This metabolic reprogramming endowed SDHA-KO CD8+ T cells with superior in vivo antitumor efficacy after immune checkpoint inhibitor therapy and allogeneic SCT. These findings reveal MC II as a bifurcation point for metabolic and functional specialization in CD4+ and CD8+ T cells.

Keywords

Animals, CD8-Positive T-Lymphocytes, Mice, CD4-Positive T-Lymphocytes, Electron Transport Complex II, Mice, Knockout, Oxidative Phosphorylation, Mitochondria, Succinate Dehydrogenase, Graft vs Host Disease, Glycolysis, Lymphocyte Activation, Hematology, Immunology, Metabolism, Bone marrow transplantation, Mitochondria, T cells

Published Open-Access

yes

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