Language

English

Publication Date

1-13-2025

Journal

Cancer Cell

DOI

10.1016/j.ccell.2024.12.006

PMID

39753140

Abstract

Glioblastoma is a highly aggressive primary brain tumor with glioblastoma stem cells (GSCs) enforcing the intra-tumoral hierarchy. Plasma cells (PCs) are critical effectors of the B-lineage immune system, but their roles in glioblastoma remain largely unexplored. Here, we leverage single-cell RNA and B cell receptor sequencing of tumor-infiltrating B-lineage cells and reveal that PCs are aberrantly enriched in the glioblastoma-infiltrating B-lineage population, experience low level of somatic hypermutation, and are associated with poor prognosis. PCs secrete immunoglobulin G (IgG), which stimulates GSC proliferation via the IgG-FcγRIIA-AKT-mTOR axis. Disruption of IgG-FcγRIIA paracrine communication inhibits GSC proliferation and self-renewal. Glioblastoma-infiltrating PCs are recruited to GSC niches via CCL2-CCR2 chemokine program. GSCs further derive pro-proliferative signals from broadly utilized monoclonal antibody-based immune checkpoint inhibitors via FcγRIIA signaling. Our data generate an atlas of B-lineage cells in glioblastoma with a framework for combinatorial targeting of both tumor cell-intrinsic and microenvironmental dependencies.

Keywords

Glioblastoma, Humans, Neoplastic Stem Cells, Immunoglobulin G, Plasma Cells, Brain Neoplasms, Animals, Mice, Tumor Microenvironment, Cell Proliferation, Receptors, IgG, Lymphocytes, Tumor-Infiltrating, Signal Transduction, Cell Line, Tumor, Receptors, CCR2, FcγRIIA, glioblastoma, glioblastoma stem cell, plasma cell, single-cell B cell receptor-seq, single-cell RNA-seq

Published Open-Access

yes

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