Language

English

Publication Date

10-1-2025

Journal

Nature Genetics

DOI

10.1038/s41588-025-02335-7

PMID

40968291

PMCID

PMC13192283

PubMedCentral® Posted Date

5-22-2026

PubMedCentral® Full Text Version

Author MSS

Abstract

Large biobanks, such as the UK Biobank (UKB), enable massive phenome by genome-wide association studies that elucidate genetic etiology of complex traits. However, individuals from diverse genetic ancestry groups are often excluded from association analyses due to concerns about population structure introducing false positive associations. Here, we generate mixed model associations and meta-analyses across genetic ancestry groups, inclusive of a larger fraction of the UKB than previous efforts, to produce freely-available summary statistics for 7,266 traits. We build a quality control and analysis framework informed by genetic architecture. Overall, we identify 14,676 significant loci (p < 5 × 10−8) in the meta-analysis that were not found in the EUR genetic ancestry group alone, including novel associations for example between CAMK2D and triglycerides. We also highlight associations from ancestry-enriched variation, including a known pleiotropic missense variant in G6PD associated with several biomarker traits. We release these results publicly alongside FAQs that describe caveats for interpretation of results, enhancing available resources for interpretation of risk variants across diverse populations.

Keywords

Humans, Genome-Wide Association Study, Biological Specimen Banks, United Kingdom, Polymorphism, Single Nucleotide, Genetic Predisposition to Disease, Phenotype, UK Biobank

Published Open-Access

yes

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