Language

English

Publication Date

1-4-2023

Journal

International Journal of Molecular Sciences

DOI

10.3390/ijms24020931

PMID

36674448

PMCID

PMC9865780

PubMedCentral® Posted Date

1-4-2023

PubMedCentral® Full Text Version

Post-print

Abstract

High-fat/sucrose diet feeding in mice causes loss of corneal nerve function and impairs corneal wound healing. While changing to a diet with a low fat/sugar composition and enrichments in complex carbohydrates mitigates the reduction in nerve function, it remains to be determined if it has an effect on corneal wound healing. In this study, 6-week-old C57BL/6 male mice were fed either a normal diet or a high-fat/sucrose diet for 20 weeks. A third group (diet reversal) was placed on a high-fat/sucrose diet for 10 weeks followed by a normal diet for an additional 10 weeks. A central corneal epithelial abrasion wound was created, and wound closure was monitored. Neutrophil and platelet recruitment was assessed by immunofluorescence microscopy. Mice fed the high-fat/sucrose diet-only had greater adiposity (p < 0.005) than normal diet-only fed mice; diet reversal markedly reduced adiposity. Following corneal abrasion, wound closure was delayed by ~6 h (p ≤ 0.01) and, at 30 h post-wounding, fewer neutrophils reached the wound center and fewer extravascular platelets were present at the limbus (p < 0.05). Diet restored normal wound closure and neutrophil and platelet influx in the injured cornea. These data suggest compositional changes to the diet may be an effective diet-based therapeutic strategy for maintaining or restoring corneal health.

Keywords

Male, Animals, Mice, Sucrose, Mice, Inbred C57BL, Cornea, Corneal Injuries, Obesity, Diet, High-Fat, cornea, macronutrient, high-fat/sucrose diet, obesity, neutrophils, platelets

Published Open-Access

yes

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