Soy Isoflavone Reduces LPS-Induced Acute Lung Injury via Increasing Aquaporin 1 and Aquaporin 5 in Rats

Authors

Yuan-Hao Lee
W Wade Kothmann
Ya-Ping Lin
Alice Z Chuang
Jeffrey S Diamond
John O'Brien

Language

English

Publication Date

8-1-2023

Journal

eNeuro

DOI

10.1523/ENEURO.0493-22.2023

PMID

37527925

PMCID

PMC10450809

PubMedCentral® Posted Date

August 2023

PubMedCentral® Full Text Version

Post-print

Abstract

Synaptic plasticity is a fundamental feature of the CNS that controls the magnitude of signal transmission between communicating cells. Many electrical synapses exhibit substantial plasticity that modulates the degree of coupling within groups of neurons, alters the fidelity of signal transmission, or even reconfigures functional circuits. In several known examples, such plasticity depends on calcium and is associated with neuronal activity. Calcium-driven signaling is known to promote potentiation of electrical synapses in fish Mauthner cells, mammalian retinal AII amacrine cells, and inferior olive neurons, and to promote depression in thalamic reticular neurons. To measure local calcium dynamics

Keywords

calcium, Connexin 36, electrical synapse, retina, SRRF

Published Open-Access

yes

Recommended Citation

Lee, Yuan-Hao; Kothmann, W Wade; Lin, Ya-Ping; et al., "Soy Isoflavone Reduces LPS-Induced Acute Lung Injury via Increasing Aquaporin 1 and Aquaporin 5 in Rats" (2023). Faculty, Staff and Student Publications. 1780.
https://digitalcommons.library.tmc.edu/uthmed_docs/1780