Faculty, Staff and Student Publications

Language

English

Publication Date

5-1-2026

Journal

Kidney Medicine

DOI

10.1016/j.xkme.2026.101326

PMID

42004656

PMCID

PMC13088993

PubMedCentral® Posted Date

3-12-2026

PubMedCentral® Full Text Version

Post-print

Abstract

Acute kidney injury (AKI) is a well-recognized complication of chimeric antigen receptor (CAR) T-cell therapy, typically attributed to cytokine release syndrome and acute tubular injury. However, glomerular disease post CAR-T cell is rare. We report a case of minimal change disease presenting with AKI and nephrotic-range proteinuria 3 weeks after B-cell maturation antigen-directed CAR-T cell therapy, ciltacabtagene autoleucel, in a patient with relapsed refractory multiple myeloma. The kidney biopsy revealed diffuse podocyte foot process effacement and a predominance of CD4+ T cells with potential on-target, off-tumor effect of B-cell maturation antigen-directed CAR-T cell therapy, leading to podocyte injury. The patient received one dose of rituximab along with a short course of corticosteroid and had complete kidney recovery by week 4 of therapy. This report emphasizes the need for further investigation into the mechanism of kidney toxicity following CAR-T cell therapy, and potential benefits and risks of immunosuppressive therapy in this context.

Keywords

CAR-T cells, multiple myeloma, glomerulonephritis, minimal change disease

Published Open-Access

yes

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