Faculty, Staff and Student Publications

Language

English

Publication Date

12-1-2024

Journal

Nature Genetics

DOI

10.1038/s41588-024-01973-7

PMID

39528826

PMCID

PMC12090064

PubMedCentral® Posted Date

12-1-2025

PubMedCentral® Full Text Version

Author MSS

Abstract

Brain metabolism perturbation can contribute to traits and diseases. We conducted a genome-wide association study for cerebrospinal fluid (CSF) and brain metabolite levels, identifying 205 independent associations (47.3% new signals, containing 11 new loci) for 139 CSF metabolites, and 32 independent associations (43.8% new signals, containing 4 new loci) for 31 brain metabolites. Of these, 96.9% (CSF) and 71.4% (brain) of the new signals belonged to previously analyzed metabolites in blood or urine. We integrated the metabolite quantitative trait loci (MQTLs) with 23 neurological, psychiatric and common human traits and diseases through colocalization to identify metabolites and biological processes implicated in these phenotypes. Combining CSF and brain, we identified 71 metabolite-trait associations, such as glycerophosphocholines with Alzheimer's disease, O-sulfo-L-tyrosine with Parkinson's disease, glycine, xanthine with waist-to-hip ratio and ergothioneine with inflammatory bowel disease. Our study expanded the knowledge of MQTLs in the central nervous system, providing insights into human traits.

Keywords

Humans, Quantitative Trait Loci, Genome-Wide Association Study, Brain, Alzheimer Disease, Polymorphism, Single Nucleotide, Xanthine, Phenotype, Glycine, Metabolome, Waist-Hip Ratio, Parkinson Disease, Tyrosine, Male

Published Open-Access

yes

Included in

Public Health Commons

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