Faculty, Staff and Student Publications

Language

English

Publication Date

10-1-2025

Journal

Alzheimer's & Dementia

DOI

10.1002/alz.70839

PMID

41152152

PMCID

PMC12567638

PubMedCentral® Posted Date

10-28-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Introduction: Mitochondrial DNA (mtDNA) variation may influence cognitive performance, but prior findings are inconsistent.

Methods: We analyzed data from 2308 participants in the Coronary Artery Risk Development in Young Adults (CARDIA) study, assessing heteroplasmy (mtHz) burden and haplogroups (mtHg) from blood-derived mtDNA at three timepoints and cognitive function across multiple domains. Multivariate linear regression models and multivariate linear mixed models adjusted for age, sex, education, and race.

Results: MtDNA heteroplasmy burden and count were not associated with cognitive performance. However, mtHgs in macro-mtHg L were linked to poorer cognitive outcomes, particularly in processing speed and global cognition in cross-sectional and longitudinal analyses. Associations remained significant after adjusting for social determinants of health (SDOH) or APOE ε4, comorbidities, and lifestyle variables.

Discussion: The mtDNA variation, especially mtHgs, may play a role in influencing cognitive trajectories, warranting further research on its role in cognitive aging.

Highlights: Heteroplasmy burden was not associated with cognitive performance at midlife. Haplogroups were associated with cognitive outcomes at midlife. Longitudinal analysis revealed that haplogroups are associated with cognitive trajectories.

Keywords

Humans, Female, Male, Cognition, DNA, Mitochondrial, Middle Aged, Haplotypes, Longitudinal Studies, Cross-Sectional Studies, Adult, Neuropsychological Tests, Cognitive Dysfunction, Mitochondria

Published Open-Access

yes

Included in

Public Health Commons

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