Language

English

Publication Date

1-1-2025

Journal

American Journal of Reproductive Immunology

DOI

10.1111/aji.70034

PMID

39739931

PMCID

PMC12743323

PubMedCentral® Posted Date

12-28-2025

PubMedCentral® Full Text Version

Author MSS

Abstract

Problem: COVID-19 during pregnancy is linked to increased maternal morbidity and a higher incidence of preterm births (PTBs), yet the underlying mechanisms remain unclear. Cellular senescence, characterized by the irreversible cessation of cell division, is a critical process in placental function, and its dysregulation has been implicated in pregnancy complications like PTB. Senescence can be induced by various stressors, including oxidative stress, DNA damage, and viral infections.

Method of study: In this study, we determined whether COVID-19 had an impact on placental senescence. We examined placentas from women infected with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) (n = 10 term, 4 preterm) compared to uninfected controls (n = 10 term, 3 preterm). The placentas were analyzed for SARS-CoV-2 infection (spike and nucleocapsid viral proteins), markers of DNA damage (γH2AX) and oxidative stress (ROS), and senescence (telomere length, cell cycle regulators, and senescence-associated secretory phenotype [SASP]).

Results: Although no overall differences in cellular senescence markers were observed between the COVID-19 positive and negative groups, we found increased secreted SASP markers. Confocal microscopy of placentas from COVID-19 positive cases revealed localized areas of oxidative stress and DNA damage colocalized with SARS-CoV-2 spike protein.

Conclusions: These findings indicate that SARS-CoV-2 infection induces localized focal placental damage, warranting further investigation into its impact on maternal and perinatal outcomes.

Keywords

Humans, Female, Pregnancy, COVID-19, Oxidative Stress, DNA Damage, Placenta, SARS-CoV-2, Pregnancy Complications, Infectious, Cellular Senescence, Adult, Spike Glycoprotein, Coronavirus, Coronavirus Infections, Histones, Betacoronavirus, Pneumonia, Viral, Phosphoproteins, Pandemics, Reactive Oxygen Species, Coronavirus Nucleocapsid Proteins, Cellular Senescence, Cell damage, COVID-19, DNA damage, Reactive Oxygen Species, SARS-CoV-2

Published Open-Access

yes

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