Language

English

Publication Date

11-1-2025

Journal

PLOS Biology

DOI

10.1371/journal.pbio.3003504

PMID

41270125

PMCID

PMC12671814

PubMedCentral® Posted Date

11-21-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Geroscience aims to target the aging process to extend healthspan. However, even isogenic individuals show heterogeneity in natural aging rate and responsiveness to pro-longevity interventions, limiting translational potential. Using RNAseq analysis of young, isogenic, subpopulations of Caenorhabditis elegans selected solely on the basis of the splicing pattern of an in vivo minigene reporter that is predictive of future life expectancy, we find a strong correlation in young animals between predicted life span and alternative splicing of mRNAs related to lipid metabolism. The activity of two RNA splicing factors, Reversed Polarity-1 (REPO-1) and Splicing Factor 1 (SFA-1), early in life is necessary for C. elegans response to specific longevity interventions and leads to context-specific changes to fat content that is mirrored by knockdown of their direct target POD-2/ACC1. Moreover, POD-2/ACC1 is required for the same longevity interventions as REPO-1/SFA-1. In addition, early inhibition of REPO-1 renders animals refractory to late onset suppression of the TORC1 pathway. Together, we propose that splicing factor activity establishes a cellular landscape early in life that enables responsiveness to specific longevity interventions and may explain variance in efficacy between individuals.

Keywords

Animals, Caenorhabditis elegans, Longevity, Caenorhabditis elegans Proteins, RNA Splicing Factors, Alternative Splicing, Lipid Metabolism, Aging

Published Open-Access

yes

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