Author ORCID Identifier

0009-0009-7833-8320

Date of Graduation

8-2025

Document Type

Thesis (MS)

Program Affiliation

Biomedical Sciences

Degree Name

Masters of Science (MS)

Advisor/Committee Chair

M. James You

Committee Member

Pamela L. Wenzel

Committee Member

Angela H. Ting

Committee Member

Shih-Han Lee

Committee Member

George A. Calin

Committee Member

Sean M. Post

Abstract

Cleavage and polyadenylation specific factor 6 (Cpsf6) translocation is frequently reported in APLL, which is Acute Promyelocytic Leukemia (APL)-like acute myeloid leukemia (AML) and other types of blood diseases associated with abnormal hematopoiesis. CPSF6 is a subunit of the Cleavage Factor I mammalian (CFIm) complex, which regulates post-transcriptional alternative polyadenylation (APA) of pre-mRNAs. Previous research indicated that Cpsf6 plays an essential role in alternative polyadenylation, development, and tumorigenesis. However, the role of Cpsf6 in hematopoiesis is unknown. Given its roles in APA, development and cancer, we hypothesize that Cpsf6 plays a critical role in hematopoiesis and that its dysregulation may have significant impacts on this process. To test this, we used conditional knockout mouse models to examine the function of Cpsf6 in both embryonic and adult hematopoiesis. Our findings demonstrate that Cpsf6 is essential for embryonic hematopoiesis but is dispensable for maintaining adult hematopoietic homeostasis.

In addition to this primary work, I contributed to related exploratory studies on RARG fusion genes reported in APLL. My efforts laid the foundation for future work on RARG fusions in leukemia.

Keywords

Hematopoiesis, CPSF6, Leukemia, APL, Alternative Polyadenylation

Available for download on Saturday, July 25, 2026

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