Author ORCID Identifier

https://orcid.org/0009-0003-1025-5330

Date of Graduation

5-2026

Document Type

Thesis (MS)

Program Affiliation

Biomedical Sciences

Degree Name

Masters of Science (MS)

Advisor/Committee Chair

Raghu Kalluri, MD, Ph.D.

Committee Member

Kunal Rai, Ph.D.

Committee Member

Scott Kopetz, MD, Ph.D.

Committee Member

Stephanie Watowich, Ph.D.

Committee Member

Xiling Shen, Ph.D.

Abstract

Colorectal cancer (CRC) is one of the most common malignancies in the United States. CRC can arise from the left or right side of the colon, and these anatomical subtypes have historically been associated with distinct developmental pathways, mutational profiles, and tumor microenvironment (TME). Left‐sided CRC (LCRC) typically follows the classical adenoma– carcinoma sequence and is characterized by recurrent mutations in APC, TP53, and KRAS, whereas many right‐sided CRC (RCRC) tumors arise from the serrated neoplasia pathway and are frequently associated with microsatellite instability (MSI) and BRAF mutations. We evaluated whether primary tumor location or microsatellite instability status is the dominant determinant of genomic and immunologic heterogeneity in CRC. We identified differentially mutated genes and T cell subtypes between tumor subgroups. While limited differences in oncogenic mutation frequencies and immune cell composition were observed between LCRC and RCRC, MSS status emerged as a stronger predictor of genomic alterations and immune phenotypes. MSI CRC primary tumors showed enrichment of oncogenic mutations in WNT signaling-associated genes, such as RNF43, and exhibited increased proportions of exhausted CD8+ T cells. In contrast, MSS CRC tumors were enriched for oncogenic mutations in APC, P53 and KRAS, and contained higher proportions of naïve CD4+ T cells. Finally, compared to MSS RNF43 WT primary tumors, MSI RNF43 Mutant primary tumors had fewer CD4+ naïve T cells. Conversely, compared to MSI RNF43 WT primary tumors, MSI RNF43 Mutant primary tumors had more CD4+ naïve T cells. Together, these findings indicate that MSS Status is a more influential predictor of an immunosuppressive TME compared to primary tumor anatomical location and MSI RNF43 Mutant CRC tumors exhibit a unique TME.

Keywords

scRNA-seq, WES, Colorectal Cancer, T-cell, Immunotherapy

Available for download on Wednesday, April 28, 2027

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