Language

English

Publication Date

1-1-2026

Journal

The FEBS Journal

DOI

10.1111/febs.70240

PMID

40875546

PMCID

PMC12820605

PubMedCentral® Posted Date

8-28-2025

PubMedCentral® Full Text Version

Post-print

Abstract

Centrosomes play a fundamental role in nucleating and organizing microtubules in the cell and are vital for faithful chromosome segregation and maintenance of genomic stability. Loss of structural or functional integrity of centrosomes causes genomic instability and is a driver of oncogenesis. Here we identify lysine demethylase 4A (KDM4A), an epigenetic 'eraser' of chromatin methyl marks, as a centrosome-localized protein, visualized at the nanometer-scale resolution. We additionally uncovered that KDM4A demethylase enzymatic activity is required to maintain centrosome homeostasis and integrity; a previously unknown functionality unlinked to altered expression of genes regulating centrosome number. We find that KDM4A interacts with and localizes to the centrosome in all stages of mitosis, where it maintains centrosome numbers and centriole engagement during mitosis. Loss of KDM4A results in supernumerary centrosomes and accrual of chromosome segregation errors including chromatin bridges and micronuclei, markers of genomic instability. In summary, these data highlight a previously unknown role for an epigenetic 'eraser' regulating centrosome integrity, mitotic fidelity, and genomic stability at the centrosome.

Keywords

Centrosome, Genomic Instability, Humans, Mitosis, Jumonji Domain-Containing Histone Demethylases, Chromosome Segregation, Chromatin, abnormal mitosis, centrosomes, genomic instability, KDM4A, mitosis

Published Open-Access

yes

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