Language

English

Publication Date

5-14-2025

Journal

Biochemical Journal

DOI

10.1042/BCJ20253103

PMID

40364616

PMCID

PMC12203960

PubMedCentral® Posted Date

5-14-2025

PubMedCentral® Full Text Version

Post-print

Abstract

3',5'-cyclic adenosine monophosphate (cAMP) is a fundamental secondary messenger capable of rapidly amplifying and propagating cellular signals in response to various extracellular stimuli. cAMP plays a significant role in hormone-mediated regulation of renal fluid and electrolyte balance. Impaired signaling of cAMP has been linked to a variety of pathological ramifications in the kidneys. This review explores the physiological functions of exchange proteins directly activated by cAMP (Epac) in renal water balance and the regulation of solute transport in the renal tubule. Additionally, the involvement of Epac signaling in renal pathologies such as acute kidney injury, chronic kidney disease, and polycystic kidney disease is discussed.

Keywords

Humans, Guanine Nucleotide Exchange Factors, Animals, Cyclic AMP, Signal Transduction, Kidney, Acute Kidney Injury, Water-Electrolyte Balance, aquaporin (AQP), electrolyte transport, epithelial Na+channel (ENaC), Na+/H+exchanger type 3 (NHE-3), polyuria

Published Open-Access

yes

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